The Cuprizone-Induced Experimental Demyelination – A Promising Animal Model for Multiple Sclerosis Type III-IV Ph.D. Thesis

نویسندگان

  • Péter Ács
  • Sámuel Komoly
چکیده

sVeto S, Acs P, Bauer J, Lassmann H, Berente Z, Sumegi B, Komoly S, Gallyas F Jr,Illes Z, 2010. Inhibiting poly(ADP-ribose) polymerase: a potential therapy againstoligodendrocyte death in multiple sclerosis. Clinical Immunology 135: S30-S30 Veto S, Acs P, Bauer J, Lassmann H, Berente Z, Sumegi B, Komoly S, Gallyas F Jr,Illes Z, 2010. Poly(ADP-ribose) polymerase (PARP) is activated in multiple sclerosis pattern III lesions and its inhibition prevents experimental demyelination andoligodendrocyte death. Journal of Neuroimmunology 222:16-16 Veto S, Acs P, Bauer J, Lassmann H, Berente Z, Sumegi B, Komoly S, Gallyas F Jr, Illes Z, 2009. Poly(ADP-ribose) polymerase (PARP) is activated in multiple sclerosispattern III lesions and its inhibition prevents experimental demyelination and

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تاریخ انتشار 2012